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Alteration of KEAP1-NRF2 activity in ALS The human brain is an organ with elevated demand of energy, relying on oxidative metabolism and high consumption of O 2 to perform its functions [68]

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Jastreboff and colleagues reported phase 2 clinical-study findings for the LY3437943 program, while earlier discovery and pharmacology data from Coskun et al

425,426,427,428 Targeting the Keap1/PGAM5/AIFM1 axis in ROS-mediated oxieptosis could selectively amplify oxidative stress to induce programmed cell death in cancer cells, offering a novel therapeutic strategy