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In mouse models of Alzheimers disease synthetic incretin hormones exerted neuroprotective effects, including reduced amyloid plaque load, reducing oxidative stress and the chronic inflammatory response in the brain, and enhancing neurogenesis (72, 73)

However, the net outcome of central stimulation leads to more prominent reductions in appetite, blood glucose and body weight in adult females, suggesting that GLP-1 receptor treatments are more effective treatments for diabetes and obesity in females compared to males

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HO is further compounded by the associated comorbidities that include endocrine dysfunction with multiple pituitary hormone deficiencies, sleep disturbance, visual dysfunction, and neurological sequalae due to the intimate colocation of the hypothalamus with other structures such as the pituitary gland, optic chiasm, and cavernous sinus [54]