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Impaired insulin signaling, diminished mitochondrial oxidative capacity, lipid peroxidation, ferroptosis, and microbial dysbiosis not only exacerbate hepatic steatosis but also facilitate the progression from simple steatosis to more severe forms of the disease, such as NASH and cirrhosis [51,56,63,64,65,66,67]

The drop in circulating leptin concentration is not exclusively caused by the change in adipose mass, as evidenced by the fact that the decline in plasma leptin was significantly larger than the change in adipose mass [123]

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