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Abstract Idiopathic Pulmonary Fibrosis (IPF) is a chronic fibrotic interstitial lung disease (ILD) of unknown etiology, characterized by increasing incidence and intricate pathogenesis

Aberrant secretion of interleukins (e.g., IL-1, IL-6) and TNF- may impair ovulatory cascade signaling, while elevated prostaglandins and dysregulated angiogenesis can interfere with follicle rupture and luteal function
Sensitive bacterial V m sensors revealed the excitability of bacterial V m and its role in antibiotic tolerance

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