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The breadth of indications reflects the peptide's capacity to act across multiple tissue types through shared mechanisms of angiogenesis, collagen synthesis, and inflammatory modulation

However, the observed phenotype in these mice was again not a simple ramification of mucosal barrier deregulation, but was the result of a conglomerate of defects in epithelial homeostasis, including epithelial cell apoptosis and barrier function irregularities

Targeting fungal redox homeostasis thus represents a promising strategy to overcome antifungal resistance

Clinical studies with large groups of subjects and controls are required to determine whether any actual link exists between ketogenic diets and disease outcomes