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On the other hand, we conclude that polarization of M1 cells during infection with highly virulent IOE promote immunopathology and tissue injury, while eliminating replicating bacteria, via multiple effector mechanisms including production of iNOS, proinflammatory cytokines, and activation of inflammatory antigen-specific Th17 cells
Abnormal neutrophil recruitment in CF airways increases oxidative stress, causing redox imbalance and elastase/IL-8 release
[DOI] [PMC free article] [PubMed] [Google Scholar] [15].Poole LB, Karplus PA, Claiborne A, Protein sulfenic acids in redox signaling, Annu
J.et al (2005a)
