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While balanced concentrations of Fe 2+ are essential for the survival of living organisms, iron overload is responsible for the generation of metabolically toxic ROS and phospholipid hydroxy peroxides through the Fenton reaction, ultimately leading to cell damage and death by ferroptosis [16, 17]
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This structural adjustment has been explored for its potential to retain certain functional properties of FOXO4 while exhibiting a longer stability profile in research settings
