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Current Knowledge and Future Directions: A Systematic Review
Timmer CJ, Sitsen JM
The therapeutic aim, therefore, is not to suppress all radical activity but to precisely modulate the redox landscape, preventing pathological accumulation that drives damage such as lipid peroxidation in disease while preserving the physiological signaling crucial for metabolic health, stress adaptation, and longevity [106, 252, 253]
It is expressed in the nucleus and is involved in the regulation of genes necessary for oligodendrocyte maturation and myelination
