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Continuous Update Project Expert Report
Long glutamate chains (~ 10 residues) can enhance the processivity of Kinesin-1 in vitro [90], whereas -tubulin polyglutamylation catalyzed by TTLL7 has been shown to inhibit Kinesin-1 motility [91]
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Wet AMD typically develops when abnormal blood vessels leak blood or fluid into the macula
