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Furthermore, as cisplatin and radiationbased treatment are likely to further select for tumors with extremely high levels of activation[18, 19, 23], ICI-based rescue of conventional treatment failure, or potentiation of conventional chemoradiation strategies may very well be unfeasible at least in a subset of HNSCC [56]

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Redox factor 1 (Ref-1) enhances specific DNA binding of p53 by promoting p53 tetramerization