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191 The mechanism of GLP-1 RA to improve dopaminergic degeneration and pathological -synuclein aggregation in the PD model may involve inhibiting the PI3K/Akt/mTOR pathway 192 and enhancing AMPK/PGC-1a signaling pathway
The medicine belongs to the GLP-1 receptor agonist class, an incretin-based treatment group that helps the body respond to rising glucose levels after meals
