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In vitro experiments proved that it reversed the phosphorylation status of IRS1, Akt, and GSK-3 and reduced beta-amyloid formation and tau hyperphosphorylation in the human neuroblastoma cell line, SH-SY5Y.31 In vivo experiments proved that liraglutide prevented the dysregulation of Akt and GSK-3 and Alzheimer-associated tau phosphorylation in the brains of diabetic mice.32,33 Besides, it prevents the loss of brain insulin receptors in an Alzheimers disease (AD) model.34 Exenatide also has a similar effect on impaired insulin signaling pathways.35,36 The GLP-1 RA and Mitochondrial Dysfunction and/or Oxidative Stress Mitochondria are the main energy production systems of most eukaryotic cells and are responsible for energy conversion, tricarboxylic acid cycle, oxidative phosphorylation, calcium storage, etc

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Female GLPDRKO mice experienced a significant increase in lean mass and maintained fat mass, leading to an overall neutral impact of cold stress on body weight gain, independent of housing temperature (Fig

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