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Trace how insights from GLP-1 physiology were translated into drug development, including key molecular engineering strategies used to optimize the stability, duration of action, and delivery of these therapeutics

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In vascular endothelium and myocardium, GLP-1R activation up-regulates eNOS via PI3KAkt, increases nitric-oxide bioavailability, and reduces NF-B-mediated inflammation, translating into improved endothelial function, lower blood pressure, and attenuation of post-infarction remodeling [14]

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