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Data show that fasting reduces DBI expression in the subpostrema border and fourth ventricle region, but treatment with exendin-4 restored expression to fed-state levels
described the activation of mammalian target of rapamycin (mTOR) as a crucial pathway for neuron protection, and that GLP-1RA affects mTOR activity by reducing caspase-3 and caspase-8 (51)
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Previous studies also demonstrated that GLP-1RAs might cross the BBB and promote bodyweight loss by acting on these nuclei
