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Tau, a microtubule-associated protein, becomes hyperphosphorylated and forms tangles inside neurons, impairing their function and leading to cell death

Pu Z, Yang Y, Qin S, Li X, Cui C, Chen W

Papaconstantinou, I

It seems in pre-clinical models that when these L-cells are exposed to perceived toxins, such as lipopolysaccharides from certain bacteria that this triggers a surge in endogenous GLP-1 which is perhaps the bodys way of using GLP-1 as an anti-inflammatory hormone or perhaps as a brake pedal on the inflammatory response in general
