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For BPC 157-treated rats, in early and in late periods, the level of oxidative stress in the liver (MDA- and NO-levels, otherwise known to be increased and critical mediators of liver fibrosis[25]) was almost continuously within normal values as it was in rats with bile duct occlusion[25]

Then, the rate-limiting enzyme in arginine synthesis, argininosuccinate synthetase 1 (ASS1), catalyzes the conversion of citrulline and aspartate into argininosuccinate, which is then cleaved into arginine and fumarate by argininosuccinate lyase (ASL) (Figure 1)

JAMA 331 , 3848 (2024)

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