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Simultaneously, when the Bcl-2/Bax imbalance disrupts the TJ proteins between alveolar epithelial cells, it directly or indirectly induces the apoptosis of alveolar epithelial cells, leading to the destruction of the alveolar epithelial barrier and increased permeability, which ultimately causes alveolar edema, alveolar collapse and refractory hypoxemia
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Here, we review the biological, biochemical, and pharmacological aspects of CD38, with emphasis on its role in NAD homeostasis
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